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Some have suggested that the aforementioned drug V-9302 targets SLC38A2 and SLC7A5 instead of SLC1A5 [34, 87] (Fig

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Figure 13D illustrates the core mechanism of this active release control

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92 Similarly, in a hamster model of pancreatic cancer, high substrate levels of glucose, linoleic acid and other fatty acids mobilized through streptozocin-induced diabetes significantly enhanced tumor growth, which was prevented by insulin treatment

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The next day, the membranes were washed and incubated with a species-specific secondary antibody for 1 hour at room temperature

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