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Accumulation of ketone bodies in the blood vessels induces neutrophilic inflammation (239)
Similar observations have been made in other disease models with high muscle glycogen, in which PGC-1 failed to promote an improvement or even worsened the outcome, e.g., Pompe disease (Takikita et al, 2010) or mice with an ablation of the mammalian target of rapamycin complex 1 (mTORC1) (Romanino et al, 2011)
We divided the study participants into two groups according to each of the nine systemic items of BVAS and compared serum GPX-3 concentrations between the two groups (Mukhtyar et al., 2009)
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