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Spectrochim Acta Part A

You don't have to tell Schulman there's an uptick in cases

Here we report the engineering and preclinical development of a bimodal molecule that integrates NMDA receptor antagonism with glucagon-like peptide-1 (GLP-1) receptor agonism to effectively reverse obesity, hyperglycaemia and dyslipidaemia in rodent models of metabolic disease

Under nutritional scarcity, DTP cells modify their metabolic processes by initiating autophagy and metabolic modification so they adjust to the hostile environment and ensure survival (88, 89)