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Packer, L.) 121126 (Academic Press, New York, New York, 1984)

Based on these findings, the reversible and short-lasting inhibition of MAO-B by safinamide administration, the absence of MAO inhibition during LJP-1207 treatment or the lack of a propargylamine moiety in both molecules, which have intrinsic neuroprotective activity [109, 110], could contribute to their ineffectiveness in our AGIHL model