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Co-factor Supplementation The use of co-factor supplementation in a wide spectrum of mitochondrial disorders has been well documented by researchers from different parts of the world [1, 7, 68, 164]

Although promising for disrupting tumor proliferation and immune evasion, STAT3 and JAK inhibitors face significant limitations due to poor BBB penetration

In our study, MT3 knockdown activated NRF2 at the stage of BMM and preosteoclasts compared with control cells

Oligomeric -synuclein binds to F0/F1-ATP synthase causing oxidation of its -subunit and also lipid peroxidation of the mitochondrial membrane, which in combination with calcium overload, leads to the formation and opening of additional mitochondrial permeability channels and, as a consequence, cell death (Ludtmann et al., 2018)