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In the FA-AKI model, FA-induced ferroptosis exacerbated renal injury by triggering inflammation to induce other types of cell death, consistent with the increase in the expression of TWEAK and Fn14 in renal tubular epithelial cells in the AKI context [164,165,166]

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Notably, 1 patient with reported sterol-27-hydroxylase (CYP27A1) deficiency, 1 patient with unclassified disease, and 7 patients with reported AKR1D1 deficiency did not have disease causing mutations identified in known BASD genes