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This corroborates with another recent study that reported sEH-dependent alleviation of social deficits and fear responses in ASD mice occurs through the restoration of dysregulated mTOR-related pathways [129]

In contrast, we identified astrocytes as the major source of all tested pro-inflammatory mediators at this early disease phase (Fig

For concentration-response linearity validation, a minimum of three parallel replicates per concentration gradient were used, a sample size commonly adopted in microbiological quantitative studies to ensure reliable linear regression analysis

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