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At the cellular level, it is theoretically speculated that androgens may upregulate TFR1 expression via AR, thereby enhancing the cells capacity for iron uptake ( 3.3 Mitochondria: the core battlefield for sex hormone regulation of ferroptosis Mitochondria are not only the primary sites of cellular energy metabolism but also major sources of reactive oxygen species (ROS) production and key hubs of iron metabolism, making them a central locus in determining whether a cell undergoes ferroptosis

Reviewing current research into which molecules show the most promise for the development of immunotherapies is an ongoing conversation that has already received a significant amount of attention (Smallwood et al., 2017

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We review cellular therapies and repurposed drugs, such as nilotinib, inosine, isradipine, iron chelators and anti-inflammatories, and discuss how their success in preclinical models has paved the way for clinical trials