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Through interdisciplinary collaboration, this approach holds promise for overcoming antibiotic resistance and microbiota disruption, ultimately contributing to safer and more effective strategies for H

When lysosomal function is severely impaired, lysosome-independent degradation pathways, such as those activated by Bortezomib, a proteasome modulator, can enhance ubiquitination and proteasomal activity, compensating for defective lysosomal function and facilitating the removal of damaged proteins and organelles, thus improving immunotherapy efficacy (133) ( Table 1 )

doi: 10.1016/j.colsurfb.2017.11.047 211 AlgahtaniMSAhmadMZAhmadJ

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