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Prolonged metabolic stress impairs mitophagy and reduces mitochondrial function, as indicated by decreased oxidative phosphorylation, impaired ATP synthesis, and elevated reactive oxygen species (ROS) generation ( Hepatic lipid overload can also directly initiate inflammatory signaling by activating resident immune cells

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In addition, thymosin beta-4s biological activities, including promotion of cell migration, angiogenesis and reduced inflammation, have been observed in human clinical trials for dermal repair, showing potential for improved recovery and reduced inflammatory responses in injured tissues