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Acknowledgment The authors gratefully acknowledge the financial support provided by Thammasat University under the TU Research scholar grant, contract number TP2/49/2556

Additionally, the S/T-3A mutation 37 , which disables serine/threonine phosphorylation sites at residues 356, 361 and 375 of XKR8, but not the 2DA mutants, enabled XKR8 KO HL-60 cells to expose PS in response to apoptosis induced by doxorubicin and NET induction by PMA, thereby restored the NET formation (Fig

[DOI] [PMC free article] [PubMed] [Google Scholar] 20.Pickart L., Vasquez-Soltero J.M., Margolina A