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Moreover, MSH3 deficiency demonstrates comprehensive therapeutic potential by correcting synaptic dysfunction, astrocytic abnormalities and locomotor deficits in HD mice ( MSH3 and PMS1 play pivotal roles in driving accelerated somatic mHTT CAG expansion rates specifically in HD-vulnerable neurons, underlying the repeat length-dependent, threshold-sensitive, selective and progressive pathogenesis in vivo

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The use of GHK results in higher glutathione (GSH) and ascorbic acid levels, better epithelialization, increased collagen synthesis, and activation of fibroblasts and mast cells in the wound

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