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DNL is regulated by key enzymes such as fatty acid synthase (FASN) and acetyl-CoA carboxylase (ACC), which are promising therapeutic targets for limiting hepatic fat accumulation [24]

319 Additionally, in aging osteoprogenitor cells, telomere attrition, DNA damage, and dysregulation of signaling pathways such as mTORC1 and Nrf2 reduce bone formation

When MT levels are sufficient, the metal is removed from the MTI, allowing it once again to bind to MTF-1, thereby inhibiting transcription (Morby et al., 1993)