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Bidirectional expression of CUG and CAG expansion transcripts and intranuclear polyglutamine inclusions in spinocerebellar ataxia type 8

Plasma and tissue GSH levels decline with age, while markers of oxidative damage increase, creating a "redox imbalance" implicated in the pathogenesis of age-related diseases [2][3][10][11]

Furthermore, the binding of DSF and copper can serve as an inhibitor for various functional proteinases related to the proteasome in multiple cancers, inducing the accumulation of essential proteins such as IKB, P27, and c-MYC, causing cell cycle arrest and triggering apoptosis (190)