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injection, as described previously 11

Moreover, ferroptotic activation induces overexpression of matrix metalloproteinases (MMPs) and a disintegrin and metalloproteinase with thrombospondin motifs (ADAMTS), which disrupts articular cartilage metabolic homeostasis and potentiates ECM degradation, thereby driving OA progression (Sheng et al., 2025)

Obviously this ignores all those who require life long treatment due to other causes of B12 deficiency (of which there are many), thereby potentially causing great harm to the vast majority of their B12 deficient patients