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19 Nitric oxide and prostaglandin E2, which caused inflammation, were inhibited with IC 50 values of 0.175 and 0.0195 M, respectively

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This distinction matters for peptide research applications

In an alkali- burn rat model, topical and systemic BPC-157 administration significantly accelerated wound closure, with histological findings demonstrating improved granulation tissue formation, faster re-epithelialisation, enhanced dermal remodelling and higher collagen deposition compared to controls.[24] In vitro assays confirmed BPC-157 enhanced proliferation and migration of human umbilical vein endothelial cells and unregulated VEGF-a expression