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Mechanisms of cancer cachexia: from metabolic dysfunction to inflammation in several target tissues In response to the high metabolic demands of cancer cells, a hyperactivation of the ubiquitinproteasome and autophagy pathways takes place resulting in compromised tissue integrity and function, particularly in skeletal muscle (Siddiqui et al., 2020)

Ingestion of specific foods affects the levels of inflammatory factors, regulates angiogenesis, modulates the estrogen metabolism pathway in the body (8), and promotes or inhibits the development of endometriosis (9)

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Volume of distribution of 0.9 L/kg