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Therapies and Models of Citrin Deficiency While the dysfunction of citrin manifests in a complex clinical phenotype across different life stages, therapeutic options remain limited, and no current treatment addresses the underlying metabolic dysregulation comprehensively

In contrast, Mdr2-/-Thy-1-/- and Mdr2-/- mice showed no differences in tumor size/numbers, although expression of HCC-associated genes AFP and GPC3, inflammation, and liver fibrosis were exacerbated in aged Mdr2-/-Thy1-/- mice (due to increased aPF activation)