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Impaired insulin signaling, diminished mitochondrial oxidative capacity, lipid peroxidation, ferroptosis, and microbial dysbiosis not only exacerbate hepatic steatosis but also facilitate the progression from simple steatosis to more severe forms of the disease, such as NASH and cirrhosis [51,56,63,64,65,66,67]

It is also involved in immune function, metabolism, tissue building and repair, sperm cell formation, and certain enzyme functions

Jiang K, Moe-Lange J, Hennet L, Feldman LJ (2016) Salt stress affects the redox status of Arabidopsis root meristems