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The use of genetic chimeras in which a fluorescently marked subset of hepatocytes was TrxR1-deficient while others were not, revealed that the multigenerational contributions of both normal and Txnrd1-deficient hepatocytes to development and to liver regeneration were indistinguishable (Rollins et al., 2010)

Efficacy of superoxide dismutase mimetic M40403 in attenuating radiation-induced oral mucositis in hamsters

Molecular detection of tumor cells in bronchoalveolar lavage fluid from patients with early stage lung cancer