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Reduced default mode network functional connectivity in patients with recurrent major depressive disorder

To optimize mitochondria-targeted therapies, enhancing drug specificity remains a priority, as existing antioxidants and iron chelators often exert systemic effects that may disrupt normal cellular functions

Chen L, Chen X-W, Huang X, Song B-L, Wang Y, Wang Y

Vitexin can not only suppress de novo lipogenesis but also enhance fatty acid oxidation and lipolysis, in addition, it can improve insulin signaling in HFD mice possibly through binding to leptin receptor and activating AMPK [33, 34]