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As consequence, the increased cellular l-2HG concentrations lead to inhibition of the electron transport and glycolysis compensating the effects of mitochondrial reductive stress induced by hypoxia (Oldham et al., 2015)

Deleting either CX3CR1 or its ligand CX3CL1 also impairs synapse elimination following sensory deprivation, such as whisker cauterization in rodents (188), further underscoring the importance of this pathway in activity dependent neural remodeling (186, 189)

Saito T, Asai K, Sato S, Hayashi M, Adachi A, Sasaki Y, et al

However, due to risks like antibiotic resistance, these approaches must be carefully managed