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We hypothesize that inhibiting intracellular glutamine transport via ASCT2, such as with V-9302, may be a more effective strategy for targeting glutamine metabolism in PIK3CA -mutant cancer cells than targeting downstream enzymatic activities, such as GLS1 or glutamine synthetase

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In some cases, further work needs to happen to find and address the root cause(s) that are contributing to the condition, such as infections , traumatic stress, and toxins

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