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The clinical and functional effects of TERT variants in myelodysplastic syndrome

Several interesting avenues for such studies have already emerged from existing literature (95)

in the other outcome, the injured tissue continues to deteriorate, and the mechanism of the persisting damage involves gliosis with the loss of support of neurotrophic factors, persistent inflammatory receptor activation, and changes in microglia and astrocytes that continue to release harmful cytokines, which promote neuronal death and axonal injury

After 20 min at RT, the fixed biofilm was washed three times for 5 min each with 2 mL of PBS, then blocked with 5% normal donkey serum (Jackson ImmunoResearch Laboratories, catalog no