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Its mechanisms involve stimulating blood vessel formation, promoting cell migration to injury sites, modulating inflammation, and interacting with nitric oxide pathways crucial for sphincter function

The slower movement of food from the stomach to the small intestine may delay the absorption of certain nutrients, such as carbohydrates and triglycerides

Given the failure of cell culture conditions to inverse the MSC phenotype following exposure to uremic circumstances, it is reasonable to hypothesize that epigenetic alterations, resembling those detected in aging MSCs, are prompted by CKD and this could potentially explain what has been referred to as uremic memory based on clinical clarifications

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