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Vitamin B12 treatment strongly affects the brain metabolome and transcriptome, and partially compensates for the effects of Tbx1 haploinsufficiency Given the ability of vB12 prenatal treatment to rescue diverse phenotypes caused by Tbx1 haploinsufficiency (13, 14, 15), we asked whether this occurs through metabolic mechanisms that are transcriptionally regulated by TBX1 or by rebalancing the brain metabolic profile of Tbx1 +/ mice

That result reframed the C-terminal region as a candidate probe for the lipid axis rather than an inert fragment

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Appropriate candidates for this treatment include people with classic TN pain in whom medications are ineffective or not tolerated, individuals with atypical features or with a diagnosis of trigeminal neuropathy, and individuals who are not deemed to be surgical candidates after review with the neurosurgical team