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Bee venom accelerates wound healing in diabetic mice by suppressing activating transcription factor-3 (ATF-3) and inducible nitric oxide synthase (iNOS)-mediated oxidative stress and recruiting bone marrow-derived endothelial progenitor cells

Therefore, although CRBN shows low affinity for dHTC1 and no detectable binding to ENL up to a concentration of 1 M, the stable, preformed ENL:( S )-dHTC1 complex can cooperatively engage CRBN with high apparent affinity to induce stereochemistry-dependent ENL degradation

FLT3 inhibitor therapy for patients with myelodysplastic syndromes (MDS) and acute myeloid leukemia (AML): impact on survival according to FLT3 status

These injuries are painful, and they almost always require emergency care to safely relocate the joint and rule out fractures or nerve damage