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Lo, M., Ling, V., Low, C., Wang, Y

Our findings provide the mechanism by which these asthma drugs attenuated the neurotoxicity of amyloid peptide, damage to heart myocytes by hypoxia-reperfusion and gentamycin-triggered kidney damage, as previously reported by several other research teams

One of the key challenges is the compensatory upregulation of other enzymes, such as TDO2, which can maintain kynurenine production in the presence of IDO1 inhibitors ( Figure 1B ) (38)