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Indeed, recent studies showed that, in addition to mediating ADCC in in vitro and ex vivo models, isatuximab and daratumumab directly activated NK cells and increased their lytic activity against tumor cells through CD16 and CD38 crosslinking [32, 33]

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[17] [18] [19] Research Literature Context Published literature on the AOD, CJC-1295, and Ipamorelin components is mixed across database records, analytical detection work, receptor or pathway research, preclinical models, and limited human-study settings