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This may be caused by insufficient ATP generation, making the astrocytes unable to cover the energetic cost of glutamate uptake, or by intracellular buildup of glutamate due to hampered oxidation

While neuronal loss following TBI occurs due to several pathological mechanisms, oxidative stresswhich is often observed following TBIis often implicated in secondary neuronal injury and death [68]

Dietary fats serve as a critical substrate for oxidative metabolism during sustained physical activity, enabling prolonged energy release and delaying glycogen depletion

Additionally, CCl4 administration increased GSSG levels, as compared to the sham groups, whereas it was significantly decreased by SAG administration (Figure 3C)