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AML LSCs show unique vulnerabilities like mitochondrial OXPHOS dependence, evidenced by studies demonstrating that dual inhibition of the glucose transporter type 1 (GLUT1) transporter and OXPHOS is required to fully eliminate LSCs in vivo [132]

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Plant Cell Rep 36:791805 Noctor G, Gomez L, Vanacker H, Foyer CH (2002) Interactions between biosynthesis, compartmentation and transport in the control of glutathione homeostasis and signalling