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Studies highlight that early in sepsis-induced acute respiratory distress syndrome (ARDS), capillary function deteriorates due to neutrophil retention, and inhibiting pro-inflammatory cytokines can mitigate ALI [27, 28, 29]

ROS generated during metabolic reprogramming (e.g., enhanced glycolysis, mitochondrial dysfunction) can amplify inflammatory signals, while inflammation further exacerbates metabolic dysregulationforming a vicious cycle of metabolic abnormalitiesinflammationimmune dysregulation (111)

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In visceral but not subcutaneous WAT, the connection between adipocyte hypertrophy and inflammation facilitated by M1-like macrophages and/or B-cells may be involved in insulin resistance [64]