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The pathogenesis of POI is complex and multifactorial and may be caused by defects in primordial follicle formation, follicle recruitment/distribution, follicle growth and development [46, 47]

In breast cancer models, both EC and ECG promoted apoptosis by upregulating pro-apoptotic proteins Bad and Bax triggering cytochrome c (cyt c) release and subsequent caspase-3 (cas-3) activation (Farhan et al