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Role of IL-6 in the regulation of neuronal development, survival and function

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doi: 10.2147/NDT.S172285 5

Muscle-specific deletions of mTOR 354 or Raptor 355 lead to progressive muscle dystrophy, diminished oxidative capacity, and glycogen accumulation, despite hyperactivation of AKT caused by disruption of mTORC1 feedback inhibition, which is mediated through S6K1IRS1 signaling