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19, 20 In this model, where NG2+ progenitor cells are a major substrate of the pathology, we found that, despite a reactive response of the O1+ oligodendroglial cells to injury, the fates of the NG2-expressing cells remain unchanged, with a similar multipotential capacity of giving rise to oligodendrocyte lineage cells, BG, and astrocytes in the cerebellum

Conclusion: Aggressive antiplatelet treatment during eCAS does not appear to reduce the risk of early stent occlusion, but neither does it increase the risk of sICH

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