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Alternatively, the biological sample may contain mRNA molecules from the test subject or genomic DNA molecules from the test subject

Mitochondrial dysfunction in arsenic toxicity Arsenic disrupts mitochondrial homeostasis through activation of the nucleotide-binding domain, leucine-rich-containing family, pyrin domain-containing-3 (NLRP3)-transforming growth factor-beta 1 (TGF-1)/Suppressors of Mothers Against Decapentaplegic (SMAD) signaling pathway, which promotes renal inflammation and fibrosis

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Studies revealed that cytochrome c leakage from mitochondria initiates apoptotic signaling cascades, ultimately resulting in cell death (Chen et al., 2006)