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However, in insulin-resistant individuals, these regulatory mechanisms are alteredresulting in increased lipolysis and the release of FFAs that accumulate in the liver and promote hepatic steatosis [105, 106]

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In the 1980s, Roediger demonstrated that butyrate serves as the primary energy source for colonocytes and that its deficiency compromises epithelial integrity and repair findings that helped establish early mechanistic concepts of the disease [28, 41]