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Control KD (siControl, ( D )) and RNF4 KD (siRNF4, ( E )) were performed in HEK BRD4-HiBiT cells for 72 h, followed by treatment with different concentrations of CCW16 (upper panel) or CCW28-3 (lower panel) for 4, 6, and 24 h

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In atherosclerotic plaque, increased matrix metalloproteinase expression and activity triggered by oxidative stress lead to its rupture and consequent thrombosis (He and Zuo, 2015)

When the NLRP3 inflammasome is activated, it activates caspase-1, which then forms active forms of pro-inflammatory cytokines such as IL-1 and IL-18 [186]