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Together, increased intraplaque cellular death and dysfunctional efferocytosis as a result of G6PD-d may accelerate plaque growth, necrotic core formation and the development of an inflammatory plaque microenvironment 106,107 (Fig

When the compound enters a cancerous cell, it becomes positively charged because water molecules replace its chloride ions

CrossRef Management of Bariatric Complications Using Endoscopic Stents: a Multi-Center Study Rena C

Conclusion: It suggests that ozone may improve oxidative stress by regulating HIF-1 and Nrf-2, reduce the oxidative stress response and inflammatory damage caused by ischemia of neuronal cells in acute cerebral infarction, thereby improving neurological function and prognosis