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During fungal infections, mitochondrial fragmentation (fission) is frequently observed, often preceding apoptotic or pyroptotic cell death ( Candida albicans infection activates DRP1, promoting mitochondrial fission and loss of mitochondrial membrane potential in epithelial cells ( In contrast, fusion events mediated by MFN1/2 can maintain mitochondrial integrity under moderate stress

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however, the residues Met1255 and Lys1256 of the FAT domain of mTOR are disordered in the intermediate state but engaged with RHEB in the active state (Fig

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