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However, activation of caspase-3 was not essential for the execution of apoptosis-like cell death of RBCs, resulting in the hypothesis that caspases were required for appropriate erythropoiesis [23]

Endothelial damage can be provoked by a number of mechanisms, such as a change in the surface tension of the plasma membrane or modification of the physicalchemical milieu of the endothelial cell through a change of intravascular pH or osmolality