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[1] [2] B12 functions as a cofactor in cellular metabolism, participating in the conversion of homocysteine to methionine and the metabolism of methylmalonic acid, both crucial for energy production

The potential explanation for sex differences in the IGF-1 signaling pathway on aging as well as response to other age-delaying interventions is unclear, but unique interactions of candidate pathways and targets with sex hormones could explain in part these disparate effects

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