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Despite such a likely connection, only one study to date has assessed differences in markers of oxidative damage in children affected with konzo, where increased levels of 8,12-iso-iPF2-VI isoprostane in affected children was associated with deficits in neurocognition, but not deficits in motor impairment 3

S-adenosylmethionine, the methyl donor utilized in all cellular methylation processes, is derived from methionine 16

Methylcobalamin provides an enhanced ability to support neurological function

More than 50% of miRNA genes are located closely within CGIs, thereby they are susceptible to other epigenetic modifications [37]