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FSP1 functions independently of GPX4 to maintain ferroptosis resistance [22,23,24]

Initial linkage analysis studies in BL/6J mice intercrossed with C3H/HeH mice revealed several genetic loci linked to glucose intolerance [14, 15], among which the inactivating mutation of Nnt contributed, with seven other loci, to ~35% of the variation of blood glucose during an IPGTT [15]

However, the interesting part of this study mentioned how as the animals aged there was no clear reduction in longevity despite the obesity that developed, but when these animals were placed on a low-fat diet, lifespan increased by 30%

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