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10.1186/s12935-022-02679-8 141 MarketM.TennakoonG.ScaffidiM.CookD

We tested this idea using genetic and pharmacological inhibition of TS activity

The findings of the study revealed that hyperoxia raised the initial amount of Nrf2 and many transcriptional targets including Immunoproteasome, 20S Proteasome, NQO1, Lon protease, and HO-1, but caused the loss of cellular capacity to adapt to signaling levels of H 2 O 2