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This exacerbates lipid peroxidation, drives excessive ROS accumulation and neuroinflammation, and ultimately triggers ferroptosis, resulting in irreversible neuronal damage and neurotoxicity (Table 1), suggesting that therapeutic targeting of ferroptosis may offer a promising strategy for the prevention and treatment of NDDs

Taken together, we speculate that these data reflect a net elevation in PCr/ATP ratio, which is considered a prognostic indicator in the progression of heart failure 40,41

Additionally, development of novel NAD+ precursors and delivery methods aims to improve bioavailability and tissue-specific targeting

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