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Increased intracellular p62 levels enable it to compete with Nrf2 for binding Keap1, resulting in Nrf2 dissociation (Taguchi et al., 2012

Inhibition of interleukin-1-stimulated MAP kinases, activating protein-1 (AP-1) and nuclear factor kappa B (NF-B) transcription factors down-regulates matrix metalloproteinase gene expression in articular chondrocytes

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