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Talwar*, Arie Croitoru, Andrew Crooks, A

By inhibiting the NLRP3 inflammasome, -HB diminishes the inflammatory microenvironment, which provides ancillary therapeutic benefits for therapeutic interventions in glioblastoma

The JNK signaling pathway is considered to be a direct mediator in PQ-induced neuronal apoptosis (Peng et al., 2004)

In addition, the human glioma TME has many immunosuppressive cells, such as M2 macrophages and Tregs, whose presence usually inhibits the activity and function of T cells and is associated with reduced overall survival of patients (179181)