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The concentrations of the reagents were optimized for quantitative NMR, ca

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Furthermore, these polysaccharides prompted intestinal IgA secretion through the activation of T cell-dependent and T cell-independent pathways, along with the stimulation and proliferation of B-cells, contributing to intestinal protection [38]

Insulin resistance promotes extracellular BCAA accumulation without altering LAT1 content, independent of prior BCAA treatment in a myotube model of skeletal muscle