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Damage caused to primary cardiomyocytes by 0.251 M of T-2 toxins was shown to trigger autophagy, and since lower autophagy levels promoted T-2-induced cytotoxicity, it was considered that autophagy could protect primary cardiomyocytes from the cytotoxicity of T-2

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doi:10.5664/jcsm.3170 Lipman, F., Dr

This metabolic switch suppresses neutrophil pulmonary infiltration by downregulating CXCR2 expression and inhibiting NADPH oxidase assembly, reducing bronchoalveolar lavage fluid (BALF) protein extravasation in septic models (Jiao et al., 2023)